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Experimental findings in mouse KD models demonstrate that reduced abundance of specific Clostridium (e.g., Clostridium sensu stricto , Clostridium clusters XIVa ) correlates significantly with vasculitis progression, whereas supplementation with these Clostridium or SCFAs alleviates vascular inflammation by either activating G protein-coupled receptors (GPR43/109A) or inhibiting HDAC activity
The safety profile was consistent with GLP-1 receptor agonists, primarily featuring transient, mild-to-moderate gastrointestinal adverse events (e.g., nausea, 55%), with low discontinuation rates (6-8.4%)
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